Androgenetic alopecia is postulated to be a dominantly inherited disorder with variable penetrance and expression. Additionally, prostate cancer, disorders of insulin resistance, and high blood pressure have been related to androgenetic alopecia.
Androgenetic alopecia is an extremely common disorder affecting both men and women. This is essentially a cosmetic disorder. A variety of genetic and environmental factors likely play a role in causing androgenetic alopecia. Other than affecting the patient psychologically, the disorder is significant only in that it allows ultraviolet light to reach the scalp and, thus, increases the amount of actinic damage. Androgenetic alopecia is a genetically determined condition. Androgen is necessary for progression of the disorder, as it is not found in males castrated prior to puberty. The progression of the disorder is stopped if postpubertal males are castrated.
At people, this condition has been also called male-pattern baldness. The hair is lost in a clearly outlined pattern, which starts above both temples. The incidence is generally considered to be greater in males than females, although some evidence suggests that the apparent differences in incidence may be a reflection of different expression in males and females. Males with androgenetic alopecia may have an increased incidence of myocardial infarction. An increase in benign prostatic hypertrophy has also been associated. If these associations are proven conclusively, this disorder will be of greater clinical significance. Androgens are important for normal male sexual development before birth and during puberty. Androgens also have other important functions in both males and females, such as regulating hair growth and sex drive.
In both males and females with androgenetic alopecia, the passage of large, dikke, with pigment coloured end-eindharen to thinner, shorter, indefinite haren and definitively the short, wispy, without pigment vellusharen in the involved areas is gradual. As the disorder progresses, the anagen phase shortens with the telogen phase remaining constant. Women with androgenetic alopecia generally lose hair diffusely over the crown. This produces a gradual thinning of the hair rather than an area of marked baldness. The part is widest anteriorly. The frontal hairline is often preserved in women with this disorder, whereas men note a gradual recession of the frontal hairline early in the process. Androgenetic alopecia in men has been associated with several other medical conditions including coronary heart disease and enlargement of the prostate, a walnut-sized gland in males that is located below the bladder.
There is no treatment for hereditary its loss, but there are three types medical treatment which can be for some people, or only or in combination useful: minoxidil, finasteride, and surgery. While most people with male pattern baldness choose to accept the condition as they accepted their hair color or shape, there are baldness treatments which can reduce or halt hair loss, and in early stages or rare cases, reverse it entirely. Minoxidil is an over-the-counter drug approved by the FDA for stimulating new hair growth and preventing further hair loss in cases of hereditary balding. Oral finasteride is an FDA approved drug for baldness and the only one available in pill form. Hair transplantation is expensive, time consuming, and sometimes painful. Hair transplantation of permanent hairs from the back and sides of the scalp to balding areas in the front is a successful procedure.
Beauty tips for men
Make sure to use after shave. It is important that you don’t abuse your skin when trying to get a close shave. After shaving, apply an oil-free moisturizer to soothe and defend the face.What is Whiteheads ? Treatment and Prevention Tips
Whiteheads can arise anywhere on the skin and are prone around eye area and cheeks. As the surface is worn away, the small white bumps) may resolve on its own.Nail Care Tips
The nail cares help protect the ends of the fingers and toes from trauma and also help us pick up small objects.