Cerebral infarction refers to the destruction of corresponding brain issue after artery occlusion. It may be associated with bloods. The pathogenesis is the formations of thrombosis or embolism.
Nature of the symptoms varies due to different blood vessels. Cerebral infarction is mainly divided into thrombosis and embolism types. The cause of non-embolic cerebral infarction is the atherosclerosis. Within the arterial wall, there is thrombosis on the basis of atherosclerotic plaque. And the cerebral artery inflammatory changes can make the blood vessel wall changes. The stenosis of lumen may produce thrombus. High blood pressure can cause hyaline degeneration of arterial wall and arterial intimal rupture. It makes the platelet easy to attach and gather thus the thrombosis forms. Polycythemia psychosis also leads to blood clots. If the cerebral blood vessels are oppressed by the tumor and other factors, the occlusive vascular changes may occur.
Embolic cerebral infarction is often the cause of blood flow into the intracranial solid, liquid, or gas emboli cerebral blood vessel blockages. Its etiology, mainly cardiac and non cardiac two categories: acute or subacute endocarditis, generally occurs on the basis of a heart attack. Due to inflammation in the lesion intima forming vegetation, with the bulky, after falling into cerebral embolism. Caused by emboli such as rheumatic heart disease, myocardial infarction, congenital heart disease, cardiac tumors, cardiac surgery, and so easy to fall off. In particular, these heart diseases, atrial fibrillation more likely to be emboli shedding can be caused by cerebral embolism. Gas embolism, long bone fractures, fat embolism, pulmonary vein thrombosis, cerebral venous thrombosis the reason of cerebral embolism. Some of finding out the source of emboli is called a cerebral infarction of unknown origin.
There were two way in past: one of them is to extend the use of thrombolytic enzyme used in about 10 days time. Study of cosmetic raw material supplier shows excessive use of thrombolytic enzymes is likely to lead to cerebral hemorrhage. The cerebral infarction patients are hypertension, atherosclerosis sclerosis and vascular weak, anti-platelet aggregation drugs such as aspirin, pan centimes, etc. However, the hazards of long-term use of these drugs are to cause coagulation disorders. The so-called coagulation disorders performance is if you do not accidentally bump the bruising is difficult to eliminate, or wound bleeding difficult to stop bleeding. Of course, cerebral hemorrhage is difficult to stop bleeding.
Recent scientific research shows a new approach is to take a certain dose of astaxanthin can effectively prevent the recurrence of cerebral infarction, without affecting the body's clotting function, to avoid the risk of cerebral hemorrhage. Patients with hypertension can also be long-term use as a drug for the prevention of cerebral infarction. Current evidence suggests that oxidative stress and the resulting inflammation is caused by abnormal thrombosis mechanism of astaxanthin pigment has powerful antioxidant and anti-inflammatory function, therefore, can effectively inhibit the oxidative stress process. It can also control of thrombosis in the process of inflammation to a certain extent Therefore, we can effectively inhibit the formation of arterial thrombosis. It greatly reduces the risk of reinfarction.
The core mechanism of astaxanthin to prevent cerebral infarction is the effective reversal of cerebral artery atherosclerosis. Long-term use of astaxanthin may completely eliminate the risk of cerebral infarction. The astaxanthin suppliers remind that its mechanism is the significant inhibition of LDL oxidation to oxidized low-density lipoprotein. It prevents the continued development of cerebral arteriosclerosis and significantly enhances the anti-atherosclerotic lipoprotein high density lipoprotein. It excretes atherosclerosis foam cells and unstable plaques. Meanwhile, it suppressed inflammatory cytokines to prevent rupture of atherosclerotic plaque and prevents the formation of blood clots. It will prevent the formation of cerebral infarction. Scientists believe that astaxanthin will launch a third wave of preventive medicines.Source:http://www.cosprm.com
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